What Are The Most Common Causes Of Face Swelling in FSGS

There are various reasons that face swelling in FSGS may occur, and most of them require some sort of medical treatment to fix the issue. Some disorders can result in swelling of the face, such assinusitis, kidney disease, and malnutrition, though an eye stye, conjunctivitis, and a tooth abscess can also contribute to facial swelling. This is also often a symptom of an allergic reaction, at which point it is typically accompanied by a rash, difficulty breathing, and even swelling inside the mouth. Finally, some drugs can cause face swelling, including commonly used medications like aspirin, penicillin, prednisone, and the substance inside asthmainhalers.
Some diseases can result in face swelling, even when it does not seem like the face should be affected by the medical condition at all. For example, kidney disease may cause this issue, as a malfunctioning kidney can fill the body with fluids, though this is usually also accompanied by swollen hands and feet. Malnutrition can also lead to body and face swelling, though it seems counterintuitive since most people assume the body should become thinner with starvation. Additionally, some smaller issues can affect the entire face, such as a tooth abscess, which can make the cheeks become inflamed and puffy. Both sinusitis and eye infections, such as a stye or conjunctivitis, can also cause the area around the eyes to become swollen, sometimes making the whole face look puffy.
Most allergic reactions are accompanied by face swelling, and some even cause the entire inside of the mouth to swell. These symptoms often also come with difficulty breathing due to the swollen mouth, a skin rash, and a fever. Some of the most common allergens include bees, pollen, and insect bites, and pet allergies. Hay fever, contact dermatitis, and allergicrhinitis can also all result in face swelling, making it important to avoid contact with the allergen in question and get medical treatment quickly.

Some people only find out that they are allergic to certain medications when their face swells up after taking the drugs. Some of the most common types of medication that can cause this reaction include steroids, penicillin, aspirin, and corticosteroids like prednisone. Additionally, asthma medication, anticonvulsants, certain diuretics, and glucosteroids can also cause face swelling in those who are allergic to it. Fortunately, it is usually possible for doctors to treat this symptom so that the swelling goes down quickly, though it may lead to difficulty breathing or death if not treated quickly.
Through the above introduction, you must have learned more knowledge about this topic. However, our final purpose is to treat FSGS thoroughly. So if you are patient and looking for a natural treatment just now, please let us know immediately, we are bound to help you try our best. 

Fish Oil Capsules For IgA Nephropathy

What is it?
Fish oil contains omega-3 fatty acids. There are two different types of omega-3 fatty acids: EPA and DHA.
Fish oil is NOT the same as cod liver oil or halibut liver oil. These fish liver oils contain large amounts of vitamin A. Large amounts of vitamin A are not recommended for people with kidney disease.
Why did my doctor prescribe or recommend it?
You have a kidney disease called IgA Nephropathy. Most people with this condition maintain normal kidney function. However, in some patients (about 30%), kidney function may get worse over time. Your doctor (nephrologist) thinks you may be one of those people whose kidneys may worsen over the years. Fish oil therapy may prevent or slow down the worsening.
Is it Effective?
Fish oil is used to slow the progression of kidney disease in people with IgA nephropathy. While fish oil capsules may be effective in many patients, some patients do not respond to it.
How does it work?
The omega-3 fatty acids in fish oil prevent inflammation in the kidney to slow down progression of kidney disease.
How will you know if it is working?
Your blood tests may show a decrease in the creatinine level (creatinine measures kidney function). Please note that fish oil does not usually decrease the amount of protein in the urine. Certain blood pressure pills and prednisone pills are used to decrease protein in the urine.
An additional benefit is that fish oil may lower the level of fats called triglycerides in your
Are there safety concerns? 
Fish oil is generally safe and most people do not experience any side effects. Others may
notice belching, fishy-smelling breath, heartburn or diarrhea. Taking your fish oil capsules with meals can decrease these side effects.
Are there any interactions with medications?
Medications that slow blood clotting (e.g. aspirin, clopidogrel (Plavix® ), warfarin (Coumadin®)):
Fish oils may slow blood clotting. Taking fish oils along with medications that also slow clotting may increase the chances of bruising and bleeding.
Medications for high blood pressure:
Fish oils may decrease blood pressure. Taking fish oils along with medications for high blood pressure may cause your blood pressure to go too low. Fish oil also might improve your blood pressure if it is too high. Good blood pressure control is an important part of treating IgA nephropathy. Target blood pressure for people with kidney disease is 130/80mm Hg or lower.
What doses are recommended?
We recommend that you take a total of 1000-1200 mg DHA and 1800-2000 mg EPA. This means taking 5-10 capsules of fish oil each day.

Causes And Treatment Of Foamy Urine

Foamy urine is something that nearly every person will experience intermittently throughout their lifetime, and while some people will not think twice about it, other register great concern over this change. The good news is that foamy urine is almost always nothing to be very concerned about, although there are scenarios it could be an indicator of an underlying health problem. While some people think that foamy urine is caused by nothing more than a reaction to a chemical used in some toilet cleaners, which is a slim possibility in isolated cases, there is a much easier biological answer behind what causes foamy urine.
In almost all cases, foamy urine is caused by nothing more than having too much protein in your system than your body can absorb before you feel the urge to use the restroom. When there is a high concentration of protein present in your urine, the protein reacts with the surrounding air and water as discharged to cause a foamy consistency in the toilet that can in some cases be likened to that on the head on a freshly poured glass of beer.
Foamy urine is not associated with any changes in urine color, painful urination, or urgency. Usually, whatever overabundance of protein is present in the body will be passed within two normally spaced trips to the bathroom and the presence of foamy urine will no longer be an issue. As such, there is no need to panic because of this as your body will self-correct the issue without needing any assistance. If it is something that is troubling to you none-the-less, drinking a couple glasses of water to speed the interval time up between trips to the bathroom is usually a sufficient fix.
If however foamy urine is a persistent issue and there is no dietary reason for it to be so prevalent, then there could be another cause which is related to an underlying medical issue. Persistent foamy urine could indicate a problem with your gallbladder or kidneys, and if that is the case then you are best advised to seek the care of a doctor for testing and instructions on continuing care. If however your diet is rich in high protein foods like certain beans, fish, meat, and poultry, that is usually the reason you are experience issues with foamy urine and there is little need for concern.
Here, we want to recommend a natural therapy known as Micro-Chinese Medicine Osmotherapy which may help you get rid of this terrible symptom as early as possible. If you want to know more, please contact us immediately, we are very happy to help you. 

Life Expectancy For Patients With Nephrotic Syndrome

If you are a patent of Nephrotic Syndrome or you have a relative with this kind of disease, how long he or she can survive must be the very matter concerned.
According to the past statistics, it is between 2 and 20 years, but why is the interval so long? The main reason is the therapies adopted during the treatment. With only western medicine, it is normally about 2 to 10 years. If only Chinese medicine, it is 2 to 8 years. But with the combination of western medicine and Chinese medicine, patients can live for 2 to 26 years! So the superiority is very obvious. Certainly, treating time is also the key, and the curative effect will be better if patients can take immediate and proper treatment once the disease is detected.
The adopted therapy of Nephrotic syndrome is directly connected with how long you can live. As for the treatment of Nephrotic syndrome, by and large, Chinese medicine is superior to western medicine. Because western medicine often employs hormone to treat nephritises, kidney disease, and Nephrotic syndrome, which can just moderate symptoms, has obvious side-effects. It can not avoid high recurrence. Meanwhile, Chinese medicine is not so. On the one hand, it has obvious curative effect and is of low recurrence, on the other hand, it won't bring pains and toxic side-effect to patients. As a result, the therapy of Chinese medicine can be equal to some developed countries of advanced medical technology. At present, we are applying the pure Chinese medicine which is combined with the most modern medical treatment technology，emphasizing on "Micro-Chinese Medicine Osmostherapy".
With the supplement of external application of Chinese medicine and steam-heated therapy to reach the repair of renal inherent cells. This kind of therapy can decrease the side effects of western medicine successfully, repair the renal lesion, block the kidney diseasing, renovate the tissue structure of Glomerular mechanical protective screen and electrical charge protective screen, and recover the Glomerular selective filtration function to reduce the protein leaking and eliminate edema. Through this characteristic therapy we can make patients of Nephrotic syndrome regain the sound build and prolong patient life effectively.
If patients of Nephrotic syndrome want a longer life, beside receiving the active treatment, they need to avoid recurrent factors. This kind of patients should cooperate with their doctors closely and make the diagnosis as early as possible. If having available condition, patients can take renal biopsy, ascertain the pathological cause from the aspect of cell structure and receiving the targeted personal therapeutic schedule; after doctors have made the schedule, what the patients should do is to do as the doctor tell them; for taking cold and infection seriously , patients would better to take medicines with the conduct of doctors. To keep a cheerful state and good diet habit, they are significant factors for the treating in the hospital and prognosis maintainance. And these factors are related with that how long patients can live. Also patients should take proper exercises to improve their constitution, which is based on their specific condition.
Finally, if you want to prolong your life expectancy to a large extent, feel free to let us know, we will try our best to help you. 

Why Occasional Abdominal Pain In IgA Nephritis Patients

IgA nephropathy is the glomerular mesangial iga deposition mainly in glomerular disease, the common pathological changes in the glomerular mesangial proliferation, therefore, from the point of view of cytology, IgA nephropathy will mesangial cell proliferative glomerulonephritis, iga deposition.
Pathogenesis: human reason for iga nephropathy, mainly due to the mucosal immune function defects in patients with bacterial or viral upper respiratory tract infection, gastrointestinal infection or urinary tract infection as an antigen enters the body, and the body of abnormal structure iga molecules combine to form the antigen-antibody immune complexes, antigen-antibody immune complexes with the general cause of flow deposition in the glomerular mesangial area, the occurrence of the inflammatory response and damage tissues and cells of the glomerulus, resulting in iga nephropathy because. Incentive there before the onset of upper respiratory tract infection, gastrointestinal infection or urinary tract infection with these.
General clinical use of conventional treatment of hormone to block the inflammatory response, it can block the progress of the disease, so that the condition improved, but in the encounter of infection, tiredness, inflammation will occur, a round repeated harm inherent in the kidney cells, so a recurrence of the situation. At this time, the body will be some uncomfortable symptoms
Micro-oriented use of integrative medicine traditional Chinese medicine in the treatment process infiltration therapy, vasodilators, anti-inflammatory, anticoagulant, the degradation of harmful substances can not only block the inflammatory response, inhibit the inflammatory response to injury kidney inherent in cells and tissues, and deposited in the kidneys of the antigen-antibody immune complexes can be cleared, so as to achieve removal from the fundamental prerequisite for blocking the inflammatory reaction to another round of the inflammatory response, repair the damaged kidney inherent cells and restore the function of kidney cells should have. 
If you are suffering from this terrible symptom for a long time or if you are looking for a natural treatment now, feel free to let us know, we will try our best to help you. 

Related To The Diagnosis And Treatment Of FSGS

More than 95% of kidney diseases are immunological disease and FSGS is not an exception. In order to give suitable treatment, the diagnosis like this judgment is very important. So for patients with FSGS, treatment should focus on immune regulation in order to achieve a good effect.
Give more care to FSGS
In the early stage, kidney disease hardly presents a symptom to patients. So many people live with it for a long time without known its existence. When their illness condition worsens, they begin to realize the problems of kidney disease. However, if they cannot hold the best time of treatment in the early stage, their illness is hardly reversed. So in the daily life, people need to pay more care to FSGS.
How is FSGS diagnosed?
From the information above, we can know that nephrotic syndrome can be diagnosed only by the symptoms. As long as patients have these certain signs, they are patients with nephrotic syndrome. However, FSGS need a very accurate test—biopsy to see the specific situation where the glomeruli are in. From the test report of biopsy, people would be diagnosed with FSGS if less than 50% of glomeruli are scarring.
How is FSGS treated?
FSGS is a serious kidney disease that needs a comprehensive treatment in regular treatment. Usually the treatment contains:
Immunosuppressive agents to suppress immune system
Diuretics and low-salt diet help remit edema
Medications for reducing high blood pressure
Statins to lower cholesterol level
In this situation, you can try Immunotherapy which is a comprehensive treatment for patients and is very considerate to improve many aspects of patients. This therapy contains several methods and focus on immune regulation to achieve a good effect.
From many patients’ experience, they only have received Immunodepression in order to block the renal inflammation. At the end, FSGS relapses repetitively. So immunotherapy adds immune regulation and immune protection to help remove renal inflammation and save kidneys from the root. These two therapies adopt Micro-Chinese Medicine Osmotherapy and Immunotherapy respectively to achieve the same aim which is to keep the immune system well and eliminate renal inflammation.
Do you have any other questions about this topic, please contact us immediately. Best wishes!

What Causes A Metallic Taste In The Mouth For FSGS

We tend to take our sense of taste for granted—until something goes wrong. A metallic taste in the mouth is a form of dysgeusia, an abnormality of the sense of taste. There are many possible causes of having a metallic taste in your mouth, ranging from medicines to systemic disorders to environmental hazards.
Medications
Medications are a leading cause of a metallic taste in the mouth for FSGS patients. The types of drugs that can cause this side effect include certain drugs that treat a wide range of conditions including bacterial infection; heart and blood pressure problems; cancer; hyperthyroid; arthritis; diabetes; heartburn; glaucoma; osteoporosis; and insomnia -- among others.
Systemic Diseases
Many systemic diseases can cause a metallic taste in the mouth, including cancer, hyperparathyroidism, renal failure, diabetes, Sjögren's syndrome, sarcoidosis, amyloidosis, vitamin B-12 deficiency and zinc deficiency.
Oral Health and Sinus Problems
Problems with the mouth and teeth can alter your sense of taste. Plaque buildup, gingivitis, periodontitis, tooth decay and abscesses can produce a bad taste in your mouth, which may be experienced as a metallic taste. Sinus infections and medicines or illnesses that cause dryness of the mouth, such as lichen planus and sicca syndrome, can also sometimes lead to a metallic taste. Similarly, dry mouth that accompanies the aging process may lead to taste disturbances.
Pregnancy
The hormone fluctuations that accompany pregnancy are believed to cause the metallic taste reported by some women, especially during the first trimester.
Nervous System Diseases
Nervous system disorders can potentially cause taste disturbances. The problem may be located in the brain or anywhere along the peripheral nerves that control the senses of taste and smell. Because taste and smell complement one another, a disturbance in your sense of smell may be perceived as an abnormality in your sense of taste. Tumors, inflammatory diseases and autoimmune diseases affecting the central or peripheral nervous system can be associated with a metallic taste in the mouth.
Allergies
Allergies have been known to cause a metallic taste in the mouth. This can be due to direct local effects on taste and smell (such as a runny or stuffy nose) or the release of histamines caused by an allergic reaction.
Metal Overload

Abnormally high levels of metals in the body, such as copper and iron, are associated with a persistent metallic taste.
Are you interested in this topic or want to know more, please contact us as early as possible, we are glad to help you. 

Treatment And Prognosis Of IgA Nephropathy

Introduction
IgA nephropathy is the most common cause of primary (idiopathic) glomerulonephritis in the developed world.Although this disorder was initially thought to follow a benign course, it is now recognized that slow progression to end-stage renal disease occurs in up to 50 percent of affected patients often over 20 to 25 years of observation. The remaining patients enter a sustained clinical remission or have persistent low grade hematuria and/or proteinuria. The prognosis is difficult to predict with accuracy in individual patients, but important risk factors for progressive renal disease have been identified.
There are two major clinical presentations of IgA nephropathy: the classic presentation with gross hematuria, often recurrent, following shortly after an upper respiratory infection; and persistent asymptomatic microscopic hematuria with or without mild to moderate proteinuria.The diagnosis may be suspected in patients with the classic presentation, but must be confirmed by kidney biopsy, which, as described below, often provides prognostic information.
The renal prognosis and treatment of IgA nephropathy will be reviewed here. The pathogenesis of IgA nephropathy and the outcomes in patients who undergo renal transplantation are discussed separately.
Renal Prognsis
Patients with IgA nephropathy who have little or no proteinuria (less than 500 to 1000mg/day) have a low risk of progression, at least in the short term. However, proteinuria and renal insufficiency develop in a substantial proportion of patients over the long term. Among patients who develop overt proteinuria and/or an elevated serum creatinine concentration, progression to end-stage renal disease is approximately 15 to 25 percent at 10 years and 20 to 30 percent at 20 years.
The rate of progression is typically slow with the GFR often falling by as little as 1 to 3 mL/min per year, a change not associated with an elevation in the serum creatinine concentration in the short term. Thus, a stable and normal serum creatinine concentration does not necessarily indicate stable disease. The frequency with which this occurs has been evaluated in studies in which repeat renal biopsy was used to assess the frequency of progressive disease. In one report, repeat renal biopsies were performed at five years in 73 patients with persistent proteinuria and a normal or near-normal initial serum creatinine. Histologic improvement occurred in only 4 percent, with 41 percent remaining stable and 55 percent showing progressive glomerular and secondary vascular and tubulointerstitial injury. An increase in serum creatinine to more than 1.5 mg/dL (133 micromol/L) was associated with major pathologic lesions.
Do you have any other questions about this topic? Please contact us as soon as possible, we will reply you within 24 hours. 

Complications of Nephrotic Syndrome

Nephrotic syndrome may be caused by primary (idiopathic) renal disease or by a variety of secondary causes. Patients present with marked edema, proteinuria, hypoalbuminemia, and often hyperlipidemia. In adults, diabetes mellitus is the most common secondary cause, and focal segmental glomerulosclerosis and membranous nephropathy are the most common primary causes. Venous thromboembolism is a possible complication; acute renal failure and serious bacterial infection are also possible, but much less common. There are no established guidelines on the diagnostic workup or management of nephrotic syndrome. Imaging studies are generally not needed, and blood tests should be used selectively to diagnose specific disorders rather than for a broad or unguided workup. Renal biopsy may be useful in some cases to confirm an underlying disease or to identify idiopathic disease that is more likely to respond to corticosteroids. Treatment of most patients should include fluid and sodium restriction, oral or intravenous diuretics, and angiotensin-converting enzyme inhibitors. Some adults with nephrotic syndrome may benefit from corticosteroid treatment, although research data are limited. Intravenous albumin, prophylactic antibiotics, and prophylactic anticoagulation are not currently recommended.
In nephrotic syndrome, a variety of disorders cause proteinuria, often resulting in marked edema and hypoalbuminemia. Hyperlipidemia is a common associated finding. Family physicians may encounter persons with nephrotic syndrome from primary (idiopathic) renal disease or a number of secondary causes, and should initiate appropriate diagnostic workup and medical management pending specialist consultation.
Excessive fluid retention: 
Massive ascites or pleural effusions may lead to respiratory compromise. Scrotal edema can result in skin breakdown and infection. Management consists of intravenous infusion of albumin along with furosemide.
Peritonitis:
Children with nephrotic syndrome are at high risk for peritonitis during the period of heavy proteinuria. The most common causative organism is Streptococcus pneumoniae; less commonly, gram-negative organisms such as E. coli are involved. Untreated peritonitis can lead to septicemia, meningitis, and death. Peritonitis should be suspected in any child with nephrotic syndrome who has abdominal pain or tenderness. Emergency paracentesis should be performed to obtain a specimen for cell count, Gram stain, and culture and broad spectrum antibiotic therapy should be immediately initiated.

Thromboembolism: 
Nephrotic syndrome is a hypercoagulable state, presumably because of loss of antithrombin-3 in the urine. Arterial and venous thrombi can develop in the major vessels, and the thrombotic tendency is potentiated by overly aggressive diuretic therapy. Sudden death may occur from massive pulmonary embolism. The index of suspicion for thrombotic episodes should be high and anticoagulation initiated immediately if a thrombus is demonstrated.
If you want to know more information about nephrotic syndrome, please contact us as soon as possible, we are here to help you. 

IgA Nephropathy Patients should Avoid Fruits And Vegetables High in Potassium

Do you know why IgA nephropathy patients should avoid fruits and vegetables high in potassium? The following may tell you some details.
One of the crucial functions of the kidneys is to maintain homeostasis, which means the body′s ability to adjust its internal environment in order to sustain life. So the kidneys can take effect to keep blood pressure, hemoglobin and blood levels of various minerals within healthy range. As IgA Nephropathy develops, the kidneys are less able to filter excessive potassium in the blood. Patients with IgA Nephropathy should avoid fruits and vegetables high in potassium.
Vegetables
Lots of vegetables are good sources of potassium, including green leafy vegetables such as broccoli, Swiss chard, mustard green, turnip greens, collard greens, kale, romaine and spinach.
Beans like lima, pinto, kidney, lentils and green beans are also high in potassium. Potatoes and cucumbers also contain high potassium, especially then we eat their skin. Other vegetables also include celery, peas, brussel sprouts, beets, asparagus, bell peppers, cauliflower, cabbage, sweet potatoes, winter and summer squashes, and eggplant.
Fruits
Fruits that are high in potassium content include bananas, all citrus fruits, cantaloupe, figs, raspberries, strawberries, plums, kiwi, grapes, apricots, tomatoes and nuts.
Some dried fruits contain higher potassium levels than fresh, such as dried apricots and prunes. They are dries plums, and one plum contains 113.52 mg of potassium and 1/4 cup of prunes contains 316.63mg.

The above are some high-potassium content fruits and vegetables that IgA Nephropathy patients should not eat. While having a proper diet of IgA Nephropathy, we need to treat the disease positively with effective treatment. Micro-Chinese Medicine Osmotherapy therapy for IgA Nephropathy is the newest therapy in 2013 in china, which develops based on many years research and clinical practices. If you are sufferering from IgA Nephropathy and want to look for a natural therapy, we are glad to help you. 

What Are the Causes of Kidneys Leaking Protein

Healthy kidneys prevent proteins in the blood stream from spilling into the urine. This is why doctors often first suspect kidney disease when a patient presents with elevated urine protein, or proteinuria. Proteinuria often occurs when the kidneys become inflamed or diseased. In a few rare instances, mild proteinuria can even be normal.
Controlling proteinuria is an important element of treatment because proteinuria itself can hasten the rate of renal failure independently of the underlying disease.
Glomerular disease
Proteinuria is often the first symptom of glomerular disease. Glomerular disease is a broad category encompassing many different diseases that affect the glomeruli. These diseases include IgA nephropathy, focal segmental glomerularsclerosis (FSGS), membranoproliferative glomerular nephritis (MPGN), membranous nephropathy, IgM nephropathy and many others. Treatment of these diseases often focuses upon controlling proteinuria and other symptoms such as hypertension.
Diabetes
Kidney disease caused by diabetes is called diabetic nephropathy. The first symptom of diabetic nephropathy is often high urine protein. Diabetic patients should have their urine checked frequently because diabetic nephropathy is the leading cause of kidney failure in the United States. According to the National Institute of Diabetes, Digestive and Kidney Diseases, 43.8 percent of all renal failure was caused by diabetes in 2007.
Orthostatic proteinuria
Mild proteinuria can be completely normal. On the website UptoDate, Dr. Burton Rose writes that 2 to 5 percent of adolescents will have orthostatic or postural proteinuria. Proteinuria is described as orthostatic if the proteinuria goes away when the patient lies down.
Birth defects
Some kidney conditions are congenital. This means that the condition is present when the baby was born. Congenital diseases that produce proteinuria are polycystic kidney disease and Alport syndrome.
Birth defects involving the kidneys do not automatically cause proteinuria. For example, proteinuria is usually absent with renal hypoplasia, the most common congenital kidney defect.
Pediatric kidney diseases
Children are more vulnerable to minimal change disease and post-streptococcal glomerular nephritis. The first symptom of both of these diseases is severe proteinuria. Although children can present multiple times with severe proteinuria due to minimal change disease, this condition usually resolves by adolescence.
Proteinuria due to post-streptococcal glomerular nephritis resolves quickly. In many instances, it is gone within several weeks.
Do you have any other questions about this topic, feel free to let us know, we are here to help you. 

Some Details About Nephrotic Syndrome In Children

Nephrotic syndrome is a condition in which your kidneys “leak” protein from your blood into your urine. In children, nephrotic syndrome may only be temporary, or it may be an early sign of kidney damage.
Are there different types of nephrotic syndrome?
Yes. The most common type of nephrotic syndrome in children is called minimal change disease. With minimal change disease, there are very tiny physical changes in the tiny filters (called glomeruli) in the kidneys. These tiny changes can affect how the kidneys work.
Minimal change disease can usually be treated with a medicine called prednisone, but this type of nephrotic syndrome can come back. This called a “relapse.” Most children will grow out of minimal change disease in their teen years.
Other types of nephrotic syndrome can be harder to treat and may cause more serious, long-term problems.
How can I keep my child from getting nephrotic syndrome?
We do not know what causes nephrotic syndrome in children, so we cannot prevent it.
What are the symptoms?
If your child has nephrotic syndrome, you may notice swelling around his or her eyes, face, ankles or feet.
How will I know if my child has nephrotic syndrome?
Your child’s doctor may need to do a few tests to find out if your child has nephrotic syndrome.
Urine test: The doctor may test a sample of your child’s urine for protein. If the first test shows a high level of protein, the doctor may want to test the urine again. You may also be asked to check your child’s urine for protein over a couple of days at home. In this case, your child’s doctor will give you directions for how to get and check the urine sample.
Blood test: The doctor may also suggest a blood test to look at the levels of electrolytes, protein, creatinine and blood urea nitrogen (BUN) in your child’s blood. These tests can help the doctor learn whether your child has nephrotic syndrome or whether there is another cause for the protein in his or her urine.
In some cases, your child’s doctor may suggest a kidney biopsy. This means that a tiny piece of your child’s kidney will be removed and viewed under a microscope. A kidney biopsy can usually be done with a needle and does not require surgery. The kidney biopsy can help show what type of nephrotic syndrome your child has and whether there are any other problems that need to be treated.
Is there a cure for nephrotic syndrome?
There is no cure for nephrotic syndrome, but many children grow out of the disease.
What are the treatments for nephrotic syndrome?
The type of treatment your child will need depends on the type of nephrotic syndrome he or she has. For minimal change disease, the most common treatment is prednisone, a type of steroid. Prednisone helps to keep protein from leaking into your child’s urine. If prednisone does not work, or if its side effects are too much, your child’s doctor may prescribe other medicines.
Other treatments may help control the swelling caused by nephrotic syndrome. A diuretic (“water pill”) can help to get rid of the extra fluid. Low-sodium (low salt) diets can help keep fluid from building up, too. In extreme cases, albumin (a kind of protein), given through an IV, can help replace some of the protein in your child’s blood.
If your child’s doctor finds any other problems while testing, those may need to be treated too.
Do you want to know some natural therapies for nephrotic syndrome? Feel free to contact us, we look forward to your consulting. 

The Proper Diet for FSGS Patients

What is the proper diet for FSGS patients? This question is consulted by more and more people. Now, follow us, you may learn more. 
That much protein and other necessary materials leak out through the urine is a very common system of FSGS (Focal Segmental Glomerulonephritis). In order to slow the worsening speed of FSGS, patients should pay more attention to their diet. A reasonable chronic FSGS diet is something that patient can easily implement.
Protein intake
In order not to increase the kidneys’ burden, patients with FSGS should control the intake of protein. However, protein is something essential our body needs, so patients should take the foods containing high quality of protein like fish, chicken, milk, lean meat and eggs and so on.
Water intake
A small amount of water is good for FSGS patients, too much fluid will be out of the damaged kidneys’ control. In order not to cause the waster retention, when patients feel thirsty, they can go for some fruits like watermelon, grapes and berries and so on.
Fat and cholesterol intake
High fat and cholesterol intake may cause many complications, so patients with FSGS should control the intake of fat and cholesterol. Egg white is good for patients, not the yolk. And foods like chocolate, candy, dessert, ice-cream, peanut, honey, butter, cream and animal fat should be controlled.
Sodium intake
In order not to aggravate high blood pressure or cause swelling, patients with FSGS should control the sodium intake. Canned foods are particularly high sources of sodium in the American diet. Fresh foods over packaged and convenience foods are recommended.
However, FSGS diet can only play a helping role, if patients want to control FSGS well or even cure it, an efficient treatment is the key. 
Finally, do you want to know some natural treatments for FSGS? If yes, please contact us as soon as possible, we will reply you immediately. 

Why Nephrotic Syndrome Is Easy To Relapse For Children

Do you know why nephrotic syndrome is easy to relapse for children? This article will give your more details. 
For children with nephrotic syndrome, after regular treatment with hormones, the symptoms such as proteinuria disappears soon, but what is depressing is that it’s easy to relapse, many parents wonder why?
There are mainly two reasons:
1. Hormones can treat symptoms but not renal damage
Without a doubt, hormones can remove symptoms of nephrotic syndrome, such as protein in urine and occult blood, in a short time. Through this kind of medication, the holes for leaking protein into urine is blocked, at the same time, some damage substance such as immune complexes is blocked in, too. It seems that the condition has turned better, but actually not, it’s just the time to hide the real problem. Under some acute conditions, hormones can be used to relieve symptoms, but they can’t be always used. It’s more important to stop the cause of renal damage and nursing kidneys.
2. Infection or cold can worsen children’s condition
The bodies of kids are not completely mature and easy to be attacked by disease, in addition, the immunity of kids with nephrotic syndrome is low and easy to get infection or cold. Infection or cold can make the burden or kidneys heavier, plus nephrotic syndrome is not throughly treated before, so it may relapse often.
If you have other questions about why nephrotic syndrome is easy to relapse for children, please contact us as soon as possible, we will try our best to help you. 

How Long Does It Take from FSGS to Kidney Failure

Do you know how long does it take from FSGS to kidney failure, the following may tell some details for you. 
FSGS is a serious kidney disorder, in which a part of glomeruli(filters) in kidneys are scarred. Some researches show that it only takes 5~10 year from FSGS to develop Kidney Failure. However, if the patients take early and proper intervention treatment along with proper health care, they will avert developing Kidney Failure.
Proteinuria and high blood pressure are two important factors in aggravating renal function deterioration. It is extremely necessary for the patients with FSGS to take medications and change diet to control these two disorders effectively.
ACEI and ARB drugs are usually prescribed to the patients. These two kinds of drugs are not only effective in lowering blood pressure, but also have certain effect in preserving the residual renal function.
Hormone shows a remarkable function in relieving protein in urine in some cases by suppressing the over-reactive immune reaction in kidney. However, it has no use in correcting the immune dysregulation and enhancing immune system.
Having a tight control of fluid and salts intake is very important in controlling blood pressure. It can reduce the blood volume so as to reduce the pressure to blood vessels.
The above measures can slow down the progression of FSGS into Kidney Failure, however, they are not capable of stopping it. Therefore, a timely and proper treatment approach should be adopted.
Immunotherapy is the sate-of-the-art therapy in treating FSGS. It is designed to treat the disease in the following ways:
It can suppress the over-reactive immune inflammatory response by using hormone and immunosuppressive drugs such as monoclonal antibody. Therefore, it can stop further damage to kidney immediately and effectively.
Immunotherapy can correct the immune dyregulation. In this way, it can prevent the triggering factors of the disease from invading into body. Thus, it can inhibit the relapse of FSGS completely.
Additionally, it can improve the renal function and boost the self-repairing of the impaired kidney cells by activating the self-curing system of the body.
A proper and effective treatment along with controlling the complications of FSGS, the patients will avert progressing into Kidney Failure.
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Treatment And Prognosis of IgA Nephropathy

IgA nephropathy is the most common cause of primary (idiopathic) glomerulonephritis in the developed world.Although this disorder was initially thought to follow a benign course, it is now recognized that slow progression to end-stage renal disease occurs in up to 50 percent of affected patients, often over 20 to 25 years of observation. The remaining patients enter a sustained clinical remission or have persistent low grade hematuria and/or proteinuria. The prognosis is difficult to predict with accuracy in individual patients, but important risk factors for progressive renal disease have been identified.
There are two major clinical presentations of IgA nephropathy: the classic presentation with gross hematuria, often recurrent, following shortly after an upper respiratory infection; and persistent asymptomatic microscopic hematuria with or without mild to moderate proteinuria.The diagnosis may be suspected in patients with the classic presentation, but must be confirmed by kidney biopsy, which, as described below, often provides prognostic information.
The renal prognosis and treatment of IgA nephropathy will be reviewed here. The pathogenesis of IgA nephropathy and the outcomes in patients who undergo renal transplantation are discussed separately.
Patients with IgA nephropathy who have little or no proteinuria (less than 500 to 1000 mg/day) have a low risk of progression, at least in the short term. However, proteinuria and renal insufficiency develop in a substantial proportion of patients over the long term.Among patients who develop overt proteinuria and/or an elevated serum creatinine concentration, progression to end-stage renal disease is approximately 15 to 25 percent at 10 years and 20 to 30 percent at 20 years.
A low antigen diet consists of avoiding gluten, dairy products, eggs, and most meats.
The rationale for this regimen is that dietary macromolecules may be responsible for activating the mucosal IgA system. When given to 21 consecutive patients with IgA nephropathy, protein excretion was markedly reduced or fell into the normal range in 11 of the 12 patients whose baseline rate was more than 1 g/day. In addition, repeat renal biopsy showed significant reductions in mesangial IgA and complement deposition and mesangial cellularity.
The benefits in the above study have not been confirmed and a report using a gluten-free diet alone for several years did not demonstrate improvement in either proteinuria or renal function despite a reduction in the level of circulating IgA-containing immune complexes
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What is Diabetic Nephropathy

Diabetic nephropathy is seen commonly in our daily life, but most people are familiar with it. The following introduces some details about this disease, a part may help you greatly. 
Signs and symptoms
Kidney failure provoked by glomerulosclerosis leads to fluid filtration deficits and other disorders of kidney function. There is an increase in blood pressure (hypertension) and fluid retention in the body plus a reduced plasma oncotic pressure causes edema. Other complications may bearteriosclerosis of the renal artery and proteinuria.
Throughout its early course, diabetic nephropathy has no symptoms. They develop in late stages and may be a result of excretion of high amounts of protein in the urine or due to renal failure:
edema: swelling, usually around the eyes in the mornings; later, general body swelling may result, such as swelling of the legs
foamy appearance or excessive frothing of the urine (caused by the proteinuria)
unintentional weight gain (from fluid accumulation)
anorexia (poor appetite)
nausea and vomiting
malaise (general ill feeling)
fatigue
headache
frequent hiccups
The first laboratory abnormality is a positive microalbuminuria test. Most often, the diagnosis is suspected when a routine urinalysis of a person with diabetes shows too much protein in the urine (proteinuria). The urinalysis may also show glucose in the urine, especially if blood glucose is poorly controlled. Serum creatinine and BUN may increase as kidney damage progresses.
A kidney biopsy confirms the diagnosis, although it is not always necessary if the case is straightforward, with a documented progression of proteinuria over time and presence of diabetic retinopathy on examination of the retina of the eyes.
Cause
The word diabetes means "passing through", referring to the polyuria (abnormal increase of urine production), a symptom historically present in those affected by the disease. When the level of blood glucose rises beyond the kidney's capacity to reabsorb glucose from the renal ultrafiltrate, glucose remains diluted in the fluid, raising its osmotic pressure and causing more water to be carried out, thus, increasing the excreted urine volume. The increased volume dilutes the sodium chloride in the urine, signalling the macula densa to release more renin, causing vasoconstriction, a survival mechanism to retain water by passing less blood through the kidneys. Because the kidney is nurtured exclusively by the blood it filtrates, the vasoconstriction also reduces the nutrients supplied to it, causing infarct of its tissues and reduction ofrenal function.
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Some Details About IgA Nephropathy

IgA nephropathy is seen commonly in our daily life, but most of people don't know what it is. the following introduces some details about IgA nephropathy, we hope a part of passage may help you greatly.
Nephropathy is damage disease, or other problems with the kidney. IgA nephropathy is a kidney disorder in which antibodies called IgA build up in kidney tissue.It is also called Berger's disease.
Causes
IgA is a protein called an antibody that helps the body fight infections. IgA nephropathy (Berger's disease) occurs when too much of this protein is deposited in the kidneys. IgA builds up inside the small blood vessels of the kidney. Structures in the kidney called glomeruli become inflamed and damaged.
IgA nephropathy (Berger's disease) is a form of mesangial proliferative nephritis.
The disorder can appear suddenly (acute), or get worse slowly over many years (chronic glomerulonephritis).
Risk factors include:
A personal or family history of IgA nephropathy or Henoch Schonlein purpura, a form of vasculitis that affects many parts of the body
Caucasian or Asian ethnicity
IgA nephropathy can occur in people of all ages, but it most often affects males in their teens to late 30s.
Symptoms
There may be no symptoms for many years.
Symptoms include:
Bloody urine that starts during or soon after a respiratory infection
Repeated episodes of dark or bloody urine
Swelling of the hands and feet
Symptoms of chronic kidney disease
Treatment
The goal of treatment is to relieve symptoms and prevent or delaychronic renal failure.
You may get medicines to control high blood pressure and swelling (edema), such as angiotensin-converting enzyme (ACE) inhibitors and angiotensin receptor blockers (ARBs). Controlling blood pressure is the most important way to delay kidney damage.
Corticosteroids, other drugs that suppress the immune system, and fish oil have also been used to treat this disorder.
Salt and fluids may be restricted to control swelling. A low to moderate protein diet may be recommended in some cases.
Some people will need to take medicines to lower their cholesterol.
Eventually, many patients must be treated for chronic kidney disease and may need dialysis.
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