IgA Nephropathy Patients should Avoid Fruits And Vegetables High in Potassium

Do you know why IgA nephropathy patients should avoid fruits and vegetables high in potassium? The following may tell you some details.
One of the crucial functions of the kidneys is to maintain homeostasis, which means the body′s ability to adjust its internal environment in order to sustain life. So the kidneys can take effect to keep blood pressure, hemoglobin and blood levels of various minerals within healthy range. As IgA Nephropathy develops, the kidneys are less able to filter excessive potassium in the blood. Patients with IgA Nephropathy should avoid fruits and vegetables high in potassium.
Vegetables
Lots of vegetables are good sources of potassium, including green leafy vegetables such as broccoli, Swiss chard, mustard green, turnip greens, collard greens, kale, romaine and spinach.
Beans like lima, pinto, kidney, lentils and green beans are also high in potassium. Potatoes and cucumbers also contain high potassium, especially then we eat their skin. Other vegetables also include celery, peas, brussel sprouts, beets, asparagus, bell peppers, cauliflower, cabbage, sweet potatoes, winter and summer squashes, and eggplant.
Fruits
Fruits that are high in potassium content include bananas, all citrus fruits, cantaloupe, figs, raspberries, strawberries, plums, kiwi, grapes, apricots, tomatoes and nuts.
Some dried fruits contain higher potassium levels than fresh, such as dried apricots and prunes. They are dries plums, and one plum contains 113.52 mg of potassium and 1/4 cup of prunes contains 316.63mg.

The above are some high-potassium content fruits and vegetables that IgA Nephropathy patients should not eat. While having a proper diet of IgA Nephropathy, we need to treat the disease positively with effective treatment. Micro-Chinese Medicine Osmotherapy therapy for IgA Nephropathy is the newest therapy in 2013 in china, which develops based on many years research and clinical practices. If you are sufferering from IgA Nephropathy and want to look for a natural therapy, we are glad to help you. 

What Are the Causes of Kidneys Leaking Protein

Healthy kidneys prevent proteins in the blood stream from spilling into the urine. This is why doctors often first suspect kidney disease when a patient presents with elevated urine protein, or proteinuria. Proteinuria often occurs when the kidneys become inflamed or diseased. In a few rare instances, mild proteinuria can even be normal.
Controlling proteinuria is an important element of treatment because proteinuria itself can hasten the rate of renal failure independently of the underlying disease.
Glomerular disease
Proteinuria is often the first symptom of glomerular disease. Glomerular disease is a broad category encompassing many different diseases that affect the glomeruli. These diseases include IgA nephropathy, focal segmental glomerularsclerosis (FSGS), membranoproliferative glomerular nephritis (MPGN), membranous nephropathy, IgM nephropathy and many others. Treatment of these diseases often focuses upon controlling proteinuria and other symptoms such as hypertension.
Diabetes
Kidney disease caused by diabetes is called diabetic nephropathy. The first symptom of diabetic nephropathy is often high urine protein. Diabetic patients should have their urine checked frequently because diabetic nephropathy is the leading cause of kidney failure in the United States. According to the National Institute of Diabetes, Digestive and Kidney Diseases, 43.8 percent of all renal failure was caused by diabetes in 2007.
Orthostatic proteinuria
Mild proteinuria can be completely normal. On the website UptoDate, Dr. Burton Rose writes that 2 to 5 percent of adolescents will have orthostatic or postural proteinuria. Proteinuria is described as orthostatic if the proteinuria goes away when the patient lies down.
Birth defects
Some kidney conditions are congenital. This means that the condition is present when the baby was born. Congenital diseases that produce proteinuria are polycystic kidney disease and Alport syndrome.
Birth defects involving the kidneys do not automatically cause proteinuria. For example, proteinuria is usually absent with renal hypoplasia, the most common congenital kidney defect.
Pediatric kidney diseases
Children are more vulnerable to minimal change disease and post-streptococcal glomerular nephritis. The first symptom of both of these diseases is severe proteinuria. Although children can present multiple times with severe proteinuria due to minimal change disease, this condition usually resolves by adolescence.
Proteinuria due to post-streptococcal glomerular nephritis resolves quickly. In many instances, it is gone within several weeks.
Do you have any other questions about this topic, feel free to let us know, we are here to help you. 

Some Details About Nephrotic Syndrome In Children

Nephrotic syndrome is a condition in which your kidneys “leak” protein from your blood into your urine. In children, nephrotic syndrome may only be temporary, or it may be an early sign of kidney damage.
Are there different types of nephrotic syndrome?
Yes. The most common type of nephrotic syndrome in children is called minimal change disease. With minimal change disease, there are very tiny physical changes in the tiny filters (called glomeruli) in the kidneys. These tiny changes can affect how the kidneys work.
Minimal change disease can usually be treated with a medicine called prednisone, but this type of nephrotic syndrome can come back. This called a “relapse.” Most children will grow out of minimal change disease in their teen years.
Other types of nephrotic syndrome can be harder to treat and may cause more serious, long-term problems.
How can I keep my child from getting nephrotic syndrome?
We do not know what causes nephrotic syndrome in children, so we cannot prevent it.
What are the symptoms?
If your child has nephrotic syndrome, you may notice swelling around his or her eyes, face, ankles or feet.
How will I know if my child has nephrotic syndrome?
Your child’s doctor may need to do a few tests to find out if your child has nephrotic syndrome.
Urine test: The doctor may test a sample of your child’s urine for protein. If the first test shows a high level of protein, the doctor may want to test the urine again. You may also be asked to check your child’s urine for protein over a couple of days at home. In this case, your child’s doctor will give you directions for how to get and check the urine sample.
Blood test: The doctor may also suggest a blood test to look at the levels of electrolytes, protein, creatinine and blood urea nitrogen (BUN) in your child’s blood. These tests can help the doctor learn whether your child has nephrotic syndrome or whether there is another cause for the protein in his or her urine.
In some cases, your child’s doctor may suggest a kidney biopsy. This means that a tiny piece of your child’s kidney will be removed and viewed under a microscope. A kidney biopsy can usually be done with a needle and does not require surgery. The kidney biopsy can help show what type of nephrotic syndrome your child has and whether there are any other problems that need to be treated.
Is there a cure for nephrotic syndrome?
There is no cure for nephrotic syndrome, but many children grow out of the disease.
What are the treatments for nephrotic syndrome?
The type of treatment your child will need depends on the type of nephrotic syndrome he or she has. For minimal change disease, the most common treatment is prednisone, a type of steroid. Prednisone helps to keep protein from leaking into your child’s urine. If prednisone does not work, or if its side effects are too much, your child’s doctor may prescribe other medicines.
Other treatments may help control the swelling caused by nephrotic syndrome. A diuretic (“water pill”) can help to get rid of the extra fluid. Low-sodium (low salt) diets can help keep fluid from building up, too. In extreme cases, albumin (a kind of protein), given through an IV, can help replace some of the protein in your child’s blood.
If your child’s doctor finds any other problems while testing, those may need to be treated too.
Do you want to know some natural therapies for nephrotic syndrome? Feel free to contact us, we look forward to your consulting. 

The Proper Diet for FSGS Patients

What is the proper diet for FSGS patients? This question is consulted by more and more people. Now, follow us, you may learn more. 
That much protein and other necessary materials leak out through the urine is a very common system of FSGS (Focal Segmental Glomerulonephritis). In order to slow the worsening speed of FSGS, patients should pay more attention to their diet. A reasonable chronic FSGS diet is something that patient can easily implement.
Protein intake
In order not to increase the kidneys’ burden, patients with FSGS should control the intake of protein. However, protein is something essential our body needs, so patients should take the foods containing high quality of protein like fish, chicken, milk, lean meat and eggs and so on.
Water intake
A small amount of water is good for FSGS patients, too much fluid will be out of the damaged kidneys’ control. In order not to cause the waster retention, when patients feel thirsty, they can go for some fruits like watermelon, grapes and berries and so on.
Fat and cholesterol intake
High fat and cholesterol intake may cause many complications, so patients with FSGS should control the intake of fat and cholesterol. Egg white is good for patients, not the yolk. And foods like chocolate, candy, dessert, ice-cream, peanut, honey, butter, cream and animal fat should be controlled.
Sodium intake
In order not to aggravate high blood pressure or cause swelling, patients with FSGS should control the sodium intake. Canned foods are particularly high sources of sodium in the American diet. Fresh foods over packaged and convenience foods are recommended.
However, FSGS diet can only play a helping role, if patients want to control FSGS well or even cure it, an efficient treatment is the key. 
Finally, do you want to know some natural treatments for FSGS? If yes, please contact us as soon as possible, we will reply you immediately. 

Why Nephrotic Syndrome Is Easy To Relapse For Children

Do you know why nephrotic syndrome is easy to relapse for children? This article will give your more details. 
For children with nephrotic syndrome, after regular treatment with hormones, the symptoms such as proteinuria disappears soon, but what is depressing is that it’s easy to relapse, many parents wonder why?
There are mainly two reasons:
1. Hormones can treat symptoms but not renal damage
Without a doubt, hormones can remove symptoms of nephrotic syndrome, such as protein in urine and occult blood, in a short time. Through this kind of medication, the holes for leaking protein into urine is blocked, at the same time, some damage substance such as immune complexes is blocked in, too. It seems that the condition has turned better, but actually not, it’s just the time to hide the real problem. Under some acute conditions, hormones can be used to relieve symptoms, but they can’t be always used. It’s more important to stop the cause of renal damage and nursing kidneys.
2. Infection or cold can worsen children’s condition
The bodies of kids are not completely mature and easy to be attacked by disease, in addition, the immunity of kids with nephrotic syndrome is low and easy to get infection or cold. Infection or cold can make the burden or kidneys heavier, plus nephrotic syndrome is not throughly treated before, so it may relapse often.
If you have other questions about why nephrotic syndrome is easy to relapse for children, please contact us as soon as possible, we will try our best to help you. 

How Long Does It Take from FSGS to Kidney Failure

Do you know how long does it take from FSGS to kidney failure, the following may tell some details for you. 
FSGS is a serious kidney disorder, in which a part of glomeruli(filters) in kidneys are scarred. Some researches show that it only takes 5~10 year from FSGS to develop Kidney Failure. However, if the patients take early and proper intervention treatment along with proper health care, they will avert developing Kidney Failure.
Proteinuria and high blood pressure are two important factors in aggravating renal function deterioration. It is extremely necessary for the patients with FSGS to take medications and change diet to control these two disorders effectively.
ACEI and ARB drugs are usually prescribed to the patients. These two kinds of drugs are not only effective in lowering blood pressure, but also have certain effect in preserving the residual renal function.
Hormone shows a remarkable function in relieving protein in urine in some cases by suppressing the over-reactive immune reaction in kidney. However, it has no use in correcting the immune dysregulation and enhancing immune system.
Having a tight control of fluid and salts intake is very important in controlling blood pressure. It can reduce the blood volume so as to reduce the pressure to blood vessels.
The above measures can slow down the progression of FSGS into Kidney Failure, however, they are not capable of stopping it. Therefore, a timely and proper treatment approach should be adopted.
Immunotherapy is the sate-of-the-art therapy in treating FSGS. It is designed to treat the disease in the following ways:
It can suppress the over-reactive immune inflammatory response by using hormone and immunosuppressive drugs such as monoclonal antibody. Therefore, it can stop further damage to kidney immediately and effectively.
Immunotherapy can correct the immune dyregulation. In this way, it can prevent the triggering factors of the disease from invading into body. Thus, it can inhibit the relapse of FSGS completely.
Additionally, it can improve the renal function and boost the self-repairing of the impaired kidney cells by activating the self-curing system of the body.
A proper and effective treatment along with controlling the complications of FSGS, the patients will avert progressing into Kidney Failure.
If you have any other questions about this topic, please contact us immediately, we will try our best to help you . 

Treatment And Prognosis of IgA Nephropathy

IgA nephropathy is the most common cause of primary (idiopathic) glomerulonephritis in the developed world.Although this disorder was initially thought to follow a benign course, it is now recognized that slow progression to end-stage renal disease occurs in up to 50 percent of affected patients, often over 20 to 25 years of observation. The remaining patients enter a sustained clinical remission or have persistent low grade hematuria and/or proteinuria. The prognosis is difficult to predict with accuracy in individual patients, but important risk factors for progressive renal disease have been identified.
There are two major clinical presentations of IgA nephropathy: the classic presentation with gross hematuria, often recurrent, following shortly after an upper respiratory infection; and persistent asymptomatic microscopic hematuria with or without mild to moderate proteinuria.The diagnosis may be suspected in patients with the classic presentation, but must be confirmed by kidney biopsy, which, as described below, often provides prognostic information.
The renal prognosis and treatment of IgA nephropathy will be reviewed here. The pathogenesis of IgA nephropathy and the outcomes in patients who undergo renal transplantation are discussed separately.
Patients with IgA nephropathy who have little or no proteinuria (less than 500 to 1000 mg/day) have a low risk of progression, at least in the short term. However, proteinuria and renal insufficiency develop in a substantial proportion of patients over the long term.Among patients who develop overt proteinuria and/or an elevated serum creatinine concentration, progression to end-stage renal disease is approximately 15 to 25 percent at 10 years and 20 to 30 percent at 20 years.
A low antigen diet consists of avoiding gluten, dairy products, eggs, and most meats.
The rationale for this regimen is that dietary macromolecules may be responsible for activating the mucosal IgA system. When given to 21 consecutive patients with IgA nephropathy, protein excretion was markedly reduced or fell into the normal range in 11 of the 12 patients whose baseline rate was more than 1 g/day. In addition, repeat renal biopsy showed significant reductions in mesangial IgA and complement deposition and mesangial cellularity.
The benefits in the above study have not been confirmed and a report using a gluten-free diet alone for several years did not demonstrate improvement in either proteinuria or renal function despite a reduction in the level of circulating IgA-containing immune complexes
If you want to know more about iga nephropathy, please contact us immediately, we will try our best to help you.